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- Figure 1.
Relationships between mutant APC signaling pathways and experimental interventions, including caloric restriction and visceral fat removal in male and female mice. AMPK, 5′ adenosine monophosphate-activated protein kinase; IKKβ-NF-κB, inhibitor of NF-κB; CXCL-1, chemokine (C-X-C motif) ligand 1; c-MYC, oncogene; ERK, extracellular signal-regulated kinase; ROS, reactive oxygen species; PKC, protein kinase C; LPL, lipoprotein lipase.
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This Perspective refers to the following article:
Abdominal Obesity, Independent from Caloric Intake, Accounts for the Development of Intestinal Tumors in Apc1638N/+ Female Mice
Derek M. Huffman, Leonard H. Augenlicht, Xueying Zhang, John J. Lofrese, Gil Atzmon, John P. Chamberland, and Christos S. Mantzoros
Volume 6, Issue 3
